Sickle Cell Anemia

Sickle Cell Anemia

Definition

Sickle Cell Anemia ek genetic blood disorder hai jisme red blood cells (RBCs) ka normal round aur flexible shape badal kar crescent ya sickle (C-shape) ho jata hai. Ye cells rigid aur fragile ho jaate hain jisse blood flow block hota hai aur oxygen transport decrease hota hai — jisse pain, anemia aur organ damage ho sakta hai.


Epidemiology

● Common in African, Indian, Middle Eastern population.

● Inherited disease — both parents ke genes se pass hota hai.

● Usually diagnosed in infancy or early childhood.


Cause

● Mutation in HBB gene on chromosome 11 → abnormal Hemoglobin S formation.


Risk Factors

● Family history of sickle cell disease.

● Both parents carrier of sickle cell trait.

● Low oxygen conditions (high altitude, dehydration, infection) trigger sickling.


Pathophysiology

● Abnormal Hemoglobin S polymerize karta hai jab oxygen kam hota hai → RBCs sickle shape me deform ho jaate hain.

● Sickled RBCs fragile hote hain → premature destruction (hemolysis) → anemia.

● Sickled cells blood vessels me chipak jaate hain → vaso-occlusion → pain crisis aur tissue ischemia.

● Chronic hypoxia → organ damage (spleen, liver, kidney, brain).


Symptoms

● Fatigue, weakness, pallor (due to anemia).

Pain crises: Sudden severe pain (chest, abdomen, joints, bones).

● Delayed growth & puberty in children.

● Jaundice (due to RBC breakdown).

● Swelling of hands and feet (dactylitis).

● Frequent infections (due to spleen damage).

● Vision problems due to retinal damage.


Complications

● Stroke (especially in children).

● Acute chest syndrome.

● Chronic organ failure (kidney, heart, spleen).

● Leg ulcers.

● Priapism (painful prolonged erection in males).


Diagnosis

● CBC → anemia with sickled RBCs.

Sickling test (positive).

● Hemoglobin electrophoresis → confirm presence of HbS.

● Genetic testing for carrier detection.


Treatment

Medical Management:

● Hydroxyurea: Increases fetal hemoglobin (HbF) → reduces sickling.

Blood transfusion: For severe anemia or crisis prevention.

Folic acid supplements: Support RBC production.

Antibiotics & vaccines: Prevent infections.

Bone marrow transplant: Only curative option (in selected cases).


Pain Crisis Management:

● Adequate hydration (IV fluids).

● Oxygen therapy.

● Analgesics (NSAIDs, opioids for severe pain).

● Monitor for infection & complications.


Nursing Management

● Monitor vital signs & oxygen saturation.

● Maintain hydration & administer fluids.

● Provide warm environment (avoid cold exposure).

● Pain assessment & management.

● Administer prescribed medications & transfusions.

● Educate patient & family:

→Avoid dehydration, stress, and infection.

→Regular follow-up & vaccination.

● Psychological support for chronic illness.


Prevention

● Genetic counseling for carrier parents.

● Avoid triggers (cold, stress, dehydration).

● Regular check-ups & medication compliance.

● Early detection & treatment of infection.


Prognosis

● Chronic but manageable disease.

● With modern treatment (Hydroxyurea, transfusion), patients can live near-normal lifespan.


Recommendation

Diabetes Mellitus Type 1

Hypertension (High Blood Pressure)

Gout-Inflammatory-Arthritis

Mycobacterium Tuberculosis

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